S. R. Prabhu - Handbook of Oral Pathology and Oral Medicine

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Handbook of Oral Pathology and Oral Medicine
Discover a concise overview of the most common oral diseases in a reader-friendly book Handbook of Oral Pathology and Oral Medicine
Handbook of Oral Pathology and Oral Medicine

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The most prevalent type of tooth wear is attrition, followed by abrasion and erosion

4.1.3 Aetiology/Risk Factors

Attrition is the physiological wearing of the tooth due to masticatory forces

Bruxism is a known cause of attrition

Abrasion is caused by abrasive dentifrices, cigars, pipes, smokeless tobacco use, improper tooth brushing techniques, and inappropriate use of dental floss or toothpicks

Erosion is caused by dietary acids from soft drinks, fruit juices and wine, chronic regurgitation of gastric contents (as in oesophageal reflux disorder, bulimia, and pregnancy), and chewable vitamin C and aspirin tablets

Abfraction occurs when occlusal forces are eccentrically applied to a tooth

4.1.4 Clinical Features

Attrition:Deciduous and permanent dentitions are affectedIncisal and occlusal surfaces are commonly affected and rarely the entire dentition may be involved ( Figure 4.1a)Other teeth may include lingual surfaces of the maxillary anterior teeth and labial surfaces of the lower anterior teethFlat large wear facets corresponding to the pattern of occlusion are commonly seenInterproximal contact points are also affectedUsually asymptomaticAttrition is a slow processDental pulp is usually protected by reactionary dentin and dentinal tubular sclerosis Figure 4.1 (a) Generalized attrition of the incisal and occlusal surfaces. (b) Abrasion of the labial surfaces of maxillary canine and premolars. (c) Erosion of the palatal surfaces in a patient with bulimia(Source: by kind permission of Associate Professor N. Narayana, UNMC Nebraska, USA.)

Abrasion:Incisal and occlusal wear is related to abrasive dietCervical wear of posterior teeth is related to faulty brushing techniquesA horizontal V‐shaped groove is commonly seen at the cervical margin of teeth in those who brush teeth vigorously or use abrasive dentifrice ( Figure 4.1b)V‐shaped notches on the incisal edges of anterior teeth are seen in those who use pipes or bobby pins, and in thread bitingAbrasion of the interproximal surfaces is seen in those who use dental floss or toothpicks inappropriately

Erosion:Palatal, occlusal, and labial surfaces of maxillary teeth are commonly affectedCommon among those with bulimia and gastroesophageal reflux disease ( Figure 4.1c)Concave facets on palatal and buccal surfaces are seenCusps are dimpledIncisal labial enamel is seen with thin, sharp, and translucent ridgeDentinal hypersensitivity is common

Abfraction:Wedge‐shaped deep defects limited to cervical area like those of abrasionOccasionally subgingival defectsFacial surfaces of premolars and molars are involved

4.1.5 Differential diagnosis

Amelogenesis imperfecta

Dentinogenesis imperfecta

Tooth defects from trauma

4.1.6 Diagnosis

History: diet, habits (clenching/bruxism), use of pipes and smokeless tobacco, tooth‐brushing techniques, regurgitation of gastric contents (bulimia, gastro‐oesophageal reflux disease), etc.

Clinical examination: location and type of wear facets and tooth defects

4.1.7 Management

Attrition: no treatment is required unless symptomatic or aesthetically unpleasant

Abrasion: restorative treatment for tooth defects to avoid abrasion

Switch to a minimally abrasive toothpaste

Erosion: removal of the cause

Fluoride application for dentinal hypersensitivity and use of a straw for soft drinks

Medical referral for those with a history of bulimia or gastro‐oesophageal reflux disease

4.1.8 Prognosis

Prognosis is good with appropriate restorative treatment

4.2 Pathological Resorption of Teeth

4.2.1 Definition/Description

Resorption of teeth:A condition associated with either a physiological or a pathological process resulting in the loss of dentin, cementum, and/or bonePhysiological resorption is a feature of shedding of deciduous teethOnly pathological resorption of permanent teeth is discussed in this chapter

External resorption:Resorption is initiated in the periodontium and initially affects the external surfaces of the toothExternal resorption may be further classified as surface, inflammatory, or replacement resorption, or by location as cervical, lateral, or apical resorption

Internal resorption:A defect of the internal aspect of the root following necrosis of odontoblasts because of chronic inflammation and bacterial invasion of the pulp tissue

4.2.2 Frequency

External resorption: common

Internal resorption: rare

4.2.3 Aetiology/Risk Factors

Causes of external resorption:Periapical periodontitisImpacted tooth pressing on the root of an adjacent tooth as evidenced on radiographyUnerupted teeth over time may show signs of resorptionReplanted teethPressure from periapical granuloma, cysts or tumoursOrthodontic treatment (common)

Causes of internal resorption:Unknown (idiopathic)

4.2.4 Clinical Features

External resorption:Asymptomatic in most casesLocalized to one tooth or a group of teethMay occur on any surface of the root and occasionally on the crown of an unerupted tooth

Internal resorption:AsymptomaticClinically, a ‘pink spot’ may be seen at the centre of the crown

4.2.5 Radiographical features

External resorption:Apex of the root is shortened ( Figure 4.2a)Opening of the apical foramen may be visibleResorbed areas may show irregular marginsRadiodensity of the resorbed area shows variation

Internal resorption:May be an incidental finding on radiographsRoot canal or pulp chamber shows enlarged radiolucent area ( Figure 4.2b)Resorbed area may be symmetrical and the walls may balloon outMargins of the resorbed area are smooth and clearly defined

4.2.6 Microscopic Features

External resorption:Numerous multinucleated dentinoclasts near the resorbed surfaceResorbed areas may show deposition of osteodentin (sign of repair)Granulation tissue in large areas of resorption

Internal resorption:Cellular and vascular fibrous connective tissueMultinucleated dentinoclastsInflammatory cells: lymphocytes, histiocytes and polymorphonuclear leukocytesPresence of woven bone as a sign of repair process Figure 4.2 Resorption. (a) External: cropped orthopantomograph shows external resorption of roots of 47 caused by impacted 48. (b) Internal: radiograph showing radiolucency in the dentinal wall of the pulp chamber of first mandibular molar.(Source: by kind permission of Dr Amar Sholapurkar, James Cook University School of Dentistry, Cairns, Australia.)

4.2.7 Differential diagnosis

Carious lesions for internal resorption and periapical lesions for external root resorption should be considered in the differential diagnosis

4.2.8 Diagnosis

History of dental procedures (orthodontic treatment in particular)

Radiography (cone beam computed tomography preferred)

4.2.9 Management

External resorption:Identification and elimination of the cause

Internal resorption:Root canal treatment

4.2.10 Prognosis

Good prognosis if the cause has been identified and eliminated and appropriate treatment is carried out

4.3 Hypercementosis

4.3.1 Definition/Description

Apposition of excess amounts of normal cementum on the root surface

Also called cemental hyperplasia

Two types occur: isolated and diffuse

4.3.2 Frequency

The prevalence of hypercementosis is not well established

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