Аланна Коллен - 10% Human. Как микробы управляют людьми

Ну вот, а пока мы — я и мои микробы — медленно возобновляем и налаживаем наши отношения. Без антибиотиков моя жизнь станет совсем другой, но теперь, когда болезнь ушла, я знаю точно одно: мои микробы должны быть на первом месте. В конце концов, я ведь человек только на 10 %.

Объем научной литературы, посвященной значению микрофлоры и микрофауны для здоровья человека, как физического, так и психического, растет в геометрической прогрессии, — ведь эту область знания начали рассматривать всерьез только в последнее десятилетие. В работе над книгой мне очень помогли личные и телефонные беседы с ведущими специалистами, электронная переписка с ними, а также рецензии, опубликованные в научных журналах. Приведенные мной сведения почерпнуты из сотен публикаций, и далеко не все они вошли в данный список литературы. В него я включила лишь наиболее важные и интересные работы, а также некоторые обобщающие труды в этой перспективной области, с которыми рекомендую читателю ознакомиться.

1. International Human Genome Sequencing Consortium (2004). Finishing the euchromatic sequence of the human genome. Nature 431: 931–945.

2. Nyholm, S.V. and McFall-Ngai, M.J. (2004). The winnowing: Establishing the squid– Vibrio symbiosis. Nature Reviews Microbiology 2: 632–642. 3. Bollinger, R.R. et al. (2007). Biofilms in the large bowel suggest an apparent function of the human vermiform appendix. Journal of Theoretical Biology 249: 826–831.

4. Short, A.R. (1947). The causation of appendicitis. British Journal of Surgery 53: 221–223.

5. Barker, D.J.P. (1985). Acute appendicitis and dietary fibre: an alternative hypothesis. British Medical Journal 290: 1125–1127.

6. Barker, D.J.P. et al. (1988). Acute appendicitis and bathrooms in three samples of British children. British Medical Journal 296: 956–958.

7. Janszky, I. et al. (2011). Childhood appendectomy, tonsillectomy, and risk for premature acute myocardial infarction — a nationwide population-based cohort study. European Heart Journal 32: 2290–2296.

8. Sanders, N.L. et al. (2013). Appendectomy and Clostridium dififcile colitis: Relationships revealed by clinical observations and immunology. World Journal of Gastroenterology 19: 5607–5614.

9. Bry, L. et al. (1996). A model of host-microbial interactions in an open mammalian ecosystem. Science 273: 1380–1383.

10. The Human Microbiome Project Consortium (2012). Structure, function and diversity of the healthy human microbiome. Nature 486: 207–214.

1. Gale, E.A.M. (2002). The rise of childhood type 1 diabetes in the 20th century. Diabetes 51: 3353–3361.

2. World Health Organisation (2014). Global Health Observatory Data — Overweight and Obesity. Available at: http://www.who.int/gho/ncd/risk_factors/overweight/en/.

3. Centers for Disease Control and Prevention (2014). Prevalence of Autism Spectrum Disorder Among Children Aged 8 Years — Autism and Developmental Disabilities Monitoring Network, 11 Sites, United States, 2010. MMWR 63 (No. SS-02): 1–21.

4. Bengmark, S. (2013). Gut microbiota, immune development and function. Pharmacological Research 69: 87–113.

5. von Mutius, E. et al. (1994) Prevalence of asthma and atopy in two areas of West and East Germany. American Journal of Respiratory and Critical Care Medicine 149: 358–364.

6. Aligne, C.A. et al. (2000). Risk factors for pediatric asthma: Contributions of poverty, race, and urban residence. American Journal of Respiratory and Critical Care Medicine 162: 873–877.

7. Ngo, S.T., Steyn, F.J. and McCombe, P.A. (2014). Gender differences in autoimmune disease. Frontiers in Neuroendocrinology 35: 347–369.

8. Krolewski, A.S. et al. (1987). Epidemiologic approach to the etiology of type 1 diabetes mellitus and its complications. The New England Journal of Medicine 26: 1390–1398.

9. Bach, J.-F. (2002). The effect of infections on susceptibility to autoimmune and allergic diseases. The New England Journal of Medicine 347: 911–920.

10. Uramoto, K.M. et al. (1999) Trends in the incidence and mortality of systemic lupus erythematosus, 1950–1992. Arthritis & Rheumatism 42: 46–50.

11. Alonso, A. and Hernan, M.A. (2008). Temporal trends in the incidence of multiple sclerosis: A systematic review. Neurology 71: 129–135.

12. Werner, S. et al. (2002). The incidence of atopic dermatitis in school entrants is associated with individual lifestyle factors but not with local environmental factors in Hannover, Germany. British Journal of Dermatology 147: 95–104.

1. Bairlein, F. (2002). How to get fat: nutritional mechanisms of seasonal fat accumulation in migratory songbirds. Naturwissenschaften 89: 1–10. 2. Heini, A.F. and Weinsier, R.L. (1997). Divergent trends in obesity and fat intake patterns: The American paradox. American Journal of Medicine 102: 259–264.

3. Silventoinen, K. et al. (2004). Trends in obesity and energy supply in the WHO MONICA Project. International Journal of Obesity 28: 710–718.

4. Troiano, R.P. et al . (2000). Energy and fat intakes of children and adolescents in the United States: data from the National Health and Nutrition Examination Surveys. American Journal of Clinical Nutrition 72: 1343s–1353s.

5. Prentice, A.M. and Jebb, S.A. (1995). Obesity in Britain: Gluttony or sloth? British Journal of Medicine 311: 437–439.

6. Westerterp, K.R. and Speakman, J.R. (2008). Physical activity energy expenditure has not declined since the 1980s and matches energy expenditures of wild mammals. International Journal of Obesity 32: 1256–1263.

7. World Health Organisation (2014). Global Health Observatory Data — Overweight and Obesity. Available at: http://www.who.int/gho/ncd/risk_factors/overweight/en/.

8. Speliotes, E.K. et al. (2010). Association analyses of 249,796 individuals reveal 18 new loci associated with body mass index. Nature Genetics 42: 937–948.

9. Marshall, J.K. et al. (2010). Eight year prognosis of postinfectious irritable bowel syndrome following waterborne bacterial dysentery. Gut 59: 605–611.

10. Gwee, K.-A. (2005). Irritable bowel syndrome in developing countries — a disorder of civilization or colonization? Neurogastroenterology and Motility 17: 317–324.

11. Collins, S.M. (2014). A role for the gut microbiota in IBS. Nature Reviews Gastroenterology and Hepatology 11: 497–505.

12. Jeffery, I.B. et al . (2012). An irritable bowel syndrome subtype defined by species-specific alterations in faecal microbiota. Gut 61: 997–1006.