Dario Polisano - The Italian Reset Diet

In the diagnosis of celiac disease, various types of analysis are addressed, ranging from the search for IgA and IgG anti-gliadin antibodies (gliadin is a part of gluten), anti-transglutaminase and anti-endomysium, to intestinal biopsies for the evaluation of the intestinal villi’s health state.

If the analyses metioned above turn out negative, but typical symptoms of the pathology still exist, and if one or more family members are celiacs, the next step in the procedure is a genetic analysis of the HLA DQ2 / DQ8 profile. Positive test results indicate an increased probability of manifesting the disease, but do not give a sure result, given that 40% of the population still has these genes. Therefore, according to conventional medicine, one should make do with the symptoms of the pathology and continue to consume gluten, until the pathology explodes in the intestines with the flattening of the villi. This is odd, considering that they usually give statins to diabetics, even with low cholesterol, for the prevention of atherosclerotic plaques. Prevention, apparently, is good only if we need to take drugs, but if it is a question of changing one’s dietary regimen, then it is not allowed anymore! I consider even more absurd the fact that the Mayo Clinic, one of the most prestigious American medical institutes, informs us how some patients with negative HLA DQ2 / DQ8 results, without the presence of antibodies, may present symptoms typical of celiac disease—a situation due to genetic predisposition with the association of the HLA DQ-α1 HLA DQ-β1 system in the class II region, which activates only the immune response of T lymphocytes, with a consequent lack of the formation of the specific autoantibodies of celiac disease. In summary, gluten is silently destroying a person's body, but as there is no clear laboratory data, then that person can calmly continue to poison himself. This very situation, which is presently studied and well-known in the scientific field but not widely recognized (for reasons I cannot understand), is known as gluten sensitivity. It is a syndrome characterized by multiple intestinal and/or extraintestinal symptoms, which occur shortly after consuming gluten. These symptoms improve or disappear after the elimination of gluten in subjects for whom the celiac disease diagnosis has been excluded. The first detections of this pathology took place in the 1980s, when female patients were described as having symptoms, credited to celiac disease or irritable bowel syndrome, that would at first disappear with the elimination of gluten and then reappear with its renewed intake. These women were thought to have psychological issues, and were treated with antidepressants!

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